Motoneuron Excitability and Muscle Spasms Are Regulated by 5-HT2B and 5-HT2C Receptor Activity

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Motoneuron excitability and muscle spasms are regulated by 5-HT2B and 5-HT2C receptor activity.

Immediately after spinal cord injury (SCI), a devastating paralysis results from the loss of brain stem and cortical innervation of spinal neurons that control movement, including a loss of serotonergic (5-HT) innervation of motoneurons. Over time, motoneurons recover from denervation and function autonomously, exhibiting large persistent calcium currents (Ca PICs) that both help with functiona...

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Motoneuron excitability and muscle spasms are regulated by 1 5 - HT

Motoneuron excitability and muscle spasms are regulated by 1 5-HT2B and 5-HT2C receptor activity. 2 3 Katherine C. Murray, Marilee J. Stephens, Edmund W. Ballou, Charles J. Heckman, 4 and David J. Bennett 5 Centre for Neuroscience, University of Alberta, Edmonton, Alberta, Canada; Department of Physiology, 6 Northwestern University, Chicago, IL, USA. 7 8 9 Running Title: 5-HT2B receptors on mot...

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Adrenergic receptors modulate motoneuron excitability, sensory synaptic transmission and muscle spasms after chronic spinal cord injury.

The brain stem provides most of the noradrenaline (NA) present in the spinal cord, which functions to both increase spinal motoneuron excitability and inhibit sensory afferent transmission to motoneurons (excitatory postsynaptic potentials; EPSPs). NA increases motoneuron excitability by facilitating calcium-mediated persistent inward currents (Ca PICs) that are crucial for sustained motoneuron...

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ژورنال

عنوان ژورنال: Journal of Neurophysiology

سال: 2011

ISSN: 0022-3077,1522-1598

DOI: 10.1152/jn.00774.2010